肺癌

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    美国研究人员发现，非烟民罹患肺癌普遍与一个基因相关，如果这个基因发生变异，活性降低，则罹患肺癌风险可能增高。
    这一发现有助于进一步研究肺癌靶向治疗和预测罹患肺癌危人群。
    确认基因
    研究人员从第一组DNA样本中找出44个最普遍的基因变异，再观察另外两组非吸烟者的这些基因变异。这两组研究对象中一半人已确诊为肺癌患者。
    结果发现，同样两个基因组是关键所在，它们负责“打开”和“关闭”GPC5基因。
    找到关键
    这种基因名为GPC5。梅奥诊所医学院研究人员发现，GPC5基因发生变异可能使非烟民罹患肺癌的风险增加。
    研究人员选取754名非吸烟者，提取他们的脱氧核糖核酸(DNA)样本，分析其中超过30万个DNA变体，研究影响非烟民罹患肺癌几率的基因变异。
    综合考虑研究对象的年龄、性别、种族、慢性呼吸道疾病史、吸入二手烟量和家族肺癌患病史以后，研究人员发现，两个基因组发挥关键作用。 

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==== 汉译英 ====
Non-smokers suffering from lung cancer gene was found to
    U.S. researchers found that non-smokers suffering from lung cancer was generally related to a gene-related, if the gene mutation, activity decreased, then the risk of lung cancer risk may be increased.
    This discovery will help further study of lung cancer, lung cancer targeted therapy and predict high-risk groups.
    Confirm gene
    The researchers from the first group of DNA samples to identify 44 of the most common gene mutation, and then observe the other two groups of non-smokers of these genetic variations. Both sets of study in half of patients with lung cancer had been diagnosed.
    The results showed that the same two genomes is the key, they are responsible for "Open" and "off" GPC5 genes.
    To find the key to
    This gene, called GPC5. Mayo Clinic School of Medicine researchers found that, GPC5 gene mutation may make non-smokers suffer an increased risk of lung cancer.
    The researchers selected 754 non-smokers, extract their DNA (DNA) sample, of which more than 300,000 DNA variants that affect risk of non-smokers lung cancer gene mutation.
    A comprehensive study to consider the age, sex, race, chronic respiratory disease history, the amount of passive smoking and family history of lung cancer in the future, the researchers found that the two genomes to play a key role.