肺癌

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    据国外媒体报道，美国波士顿哈佛大学医学院的研究人员通过研究，发现一种杀死癌细胞的新分子路径，这种方法不是利用有毒性很大的药物，而是采用分子途径，迫使癌细胞衰老、死亡。
    由于癌细胞会不断分裂，而且不会经历正常的衰老过程，因此会不断扩散、生长。但是对老鼠进行的一项试验显示，限制这一路径中一个被称作Skp2的基因的活性，可导致癌细胞进入衰老过程，开始停止分裂，能有效阻止肿瘤继续增长。哈佛大学医学院的皮埃尔·保罗·潘多尔菲和同事们把这项研究成果发表在《自然》杂志上。他表示，这项研究或许会为人们提供一种对抗癌症的新方法。
    当他们给实验室培养的人类前列腺癌细胞施加Skp2基因阻滞药物时，也得到了类似结果。该科研组表示，这个衰老路径显然只跟癌细胞有关，跟其他细胞没有任何联系，这说明它将有助于预防和治疗癌症。潘多尔菲和同事们写道，利用药物减小这种基因的活性，会为预防和治疗癌症提供一种“全新的‘提前衰老(pro-senescence)’疗法”。
    为了这项研究，哈佛大学的这些科学家利用患上前列腺癌的转基因老鼠进行试验。他们减弱了其中一些老鼠体内的Skp2基因的活性。当这些老鼠长到6个月大时，他们发现，那些Skp2基因不活跃的老鼠，体内形成的肿瘤明显更小。他们对从Skp2基因不活跃的老鼠的淋巴腺和前列腺里获得的组织进行分析，结果发现很多细胞已经开始衰老，他们还发现，这些老鼠体内癌细胞的分裂速度更慢一些。但他们并没在Skp2基因功能正常的老鼠身上发现上述现象。

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==== 汉译英 ====
According to foreign media reports, the U.S. researchers at Harvard Medical School in Boston, through research, discovered a new molecular pathways to kill cancer cells, this method is not used very toxic drug, but the molecular means to force cancer cells aging, death.
    As the cancer cells will continue to split and will not experience the normal aging process, it will continue to spread and grow. However, an experiment conducted on rats showed that restriction of this path is called a Skp2 gene activity that can lead to cancer cells into the aging process began to stop splitting, can effectively prevent the tumor continued to grow. Harvard Medical School Pierre-Paul Luo Pan Dolfi, and his colleagues put the results of the study, published in "Nature" magazine. He said the study may provide people with a new method to combat cancer.
    When they give laboratory cultures of human prostate cancer cells to exert Skp2 gene block drugs, also received similar results. The research group said that the path is clear only with cancer cells of aging related to no contact with other cells, indicating that it will contribute to the prevention and treatment of cancer. Pandolfi and his colleagues wrote that the use of drugs reduce the activity of this gene will be for the prevention and treatment of cancer offers a "new 'early aging (pro-senescence)' therapy."
    For this study, the scientists at Harvard University suffering from prostate cancer using genetically modified mice tested. They weaken some of the mice of the Skp2 gene activity. When these mice grew to 6 months, they found that those who Skp2 gene inactive mice, the formation of tumors in vivo significantly smaller. They are not active from the Skp2 gene in mice where the lymph glands and prostate tissues obtained for analysis, the results found that many cells have begun aging, they also found that these mice cancer cells divide more slowly some. But they did not in Skp2 gene function in normal mice was found the above phenomenon.